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We are deeply saddened by the passing of Amos Bairoch (1957–2025), the creator of PROSITE. We wish to dedicate our latest paper, published shortly before his death, to him. He will always be a source of inspiration to us.
Our deepest condolences go out to his family and friends, and to all those who had the privilege of working with him. Rest in peace, Amos. Your work will live on long after you are gone.
Amos Bairoch

PROSITE documentation PDOC51290
CRIC domain profile


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PURL: https://purl.expasy.org/prosite/documentation/PDOC51290

Description

The multidomain protein Connector enhancer of kinase suppressor of ras (Connector enhancer of KSR) (CNK) functions as a scaffold in several signal cascades and acts on proliferation, differenciation and apoptosis. CNK connects upstream activators and downstream targets of Ras- and Rho-dependent signaling pathways and may allow cross-talks between these pathways. In invertebrates, CNK is expressed as one isoform, whereas in mammals there exists CNK1, CNK2A, and its splice variant CNK2B. CNK proteins consist of one sterile α motif (SAM) domain (see <PDOC50105>), one conserved region in CNK (CRIC) domain, one PSD-96/Dlg-A/ZO-1 (PDZ) domain (see <PDOC50106>), and one pleckstrin homology (PH) domain (see <PDOC50003>). The CRIC domain is enriched in leucine residues and functions as a protein-protein interaction domain [1,2].

The profile we developed covers the entire CRIC domain.

Last update:

January 2007 / First entry.

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Technical section

PROSITE method (with tools and information) covered by this documentation:

CRIC, PS51290; CRIC domain profile  (MATRIX)


References

1AuthorsIida J. Nishimura W. Yao I. Hata Y.
TitleSynaptic localization of membrane-associated guanylate kinase-interacting protein mediated by the pleckstrin homology domain.
SourceEur. J. Neurosci. 15:1493-1498(2002).
PubMed ID12028359

2AuthorsFritz R.D. Radziwill G.
TitleThe scaffold protein CNK1 interacts with the angiotensin II type 2 receptor.
SourceBiochem. Biophys. Res. Commun. 338:1906-1912(2005).
PubMed ID16289034
DOI10.1016/j.bbrc.2005.10.168



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