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We are deeply saddened by the passing of Amos Bairoch (1957–2025), the creator of PROSITE. We wish to dedicate our latest paper, published shortly before his death, to him. He will always be a source of inspiration to us.
Our deepest condolences go out to his family and friends, and to all those who had the privilege of working with him. Rest in peace, Amos. Your work will live on long after you are gone.
Amos Bairoch

PROSITE documentation PDOC51269
COMM domain profile


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PURL: https://purl.expasy.org/prosite/documentation/PDOC51269

Description

COMM (copper metabolism gene MURR1) domain proteins constitute a family initially identified as interacting partners of COMMD1 (previously known as MURR1), the prototype member of this protein family. COMMD1 is a multifunctional protein that has been shown to participate in two apparently distinct activities, regulation of the transcription factor NF-kappa-B and control of copper metabolism. The family is defined by the presence of a C-terminal motif termed COMM domain, which functions as an interface for protein-protein interactions. The proteins designated as COMMD or COMM domain containing 1-10 are extensively conserved in multicellular eukaryotic organisms [1,2].

The leucine-rich, 70-85 amino acid long COMM domain is predicted to form a β-sheet [1].

The profile we developed covers the entire COMM domain.

Last update:

November 2006 / First entry.

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Technical section

PROSITE method (with tools and information) covered by this documentation:

COMM, PS51269; COMM domain profile  (MATRIX)


References

1AuthorsBurstein E. Hoberg J.E. Wilkinson A.S. Rumble J.M. Csomos R.A. Komarck C.M. Maine G.N. Wilkinson J.C. Mayo M.W. Duckett C.S.
TitleCOMMD proteins, a novel family of structural and functional homologs of MURR1.
SourceJ. Biol. Chem. 280:22222-22232(2005).
PubMed ID15799966
DOI10.1074/jbc.M501928200

2Authorsde Bie P. van de Sluis B. Burstein E. Duran K.J. Berger R. Duckett C.S. Wijmenga C. Klomp L.W.J.
TitleCharacterization of COMMD protein-protein interactions in NF-kappaB signalling.
SourceBiochem. J. 398:63-71(2006).
PubMed ID16573520
DOI10.1042/BJ20051664



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