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We are deeply saddened by the passing of Amos Bairoch (1957–2025), the creator of PROSITE. We wish to dedicate our latest paper, published shortly before his death, to him. He will always be a source of inspiration to us.
Our deepest condolences go out to his family and friends, and to all those who had the privilege of working with him. Rest in peace, Amos. Your work will live on long after you are gone.
Amos Bairoch

PROSITE documentation PDOC51117
Laminin N-terminal (LN) domain profile


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PURL: https://purl.expasy.org/prosite/documentation/PDOC51117

Description

Laminin is a large molecular weight glycoprotein present only in basement membranes in almost every animal tissue. Each laminin is a heterotrimer assembled from α, β and γ chain subunits, secreted and incorporated into cell-associated extracellular matrices [1].

Basement membrane assembly is a cooperative process in which laminins polymerize through their N-terminal domain (LN or domain VI) and anchor to the cell surface through their G domains (see <PDOC50025>). Netrins may also associate with this network through heterotypic LN domain interactions [2]. This leads to cell signaling through integrins and dystroglycan (and possibly other receptors) recruited to the adherent laminin. This LN domain dependent self-assembly is considered to be crucial for the integrity of basement membranes, as highlighted by genetic forms of muscular dystrophy containing the deletion of the LN module from the α 2 laminin chain [3].

The laminin N-terminal domain is found in all laminin and netrin subunits except laminin α 3A, α 4 and γ 2.

The profile we developed covers the whole laminin N-terminal domain.

Last update:

April 2005 / First entry.

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Technical section

PROSITE method (with tools and information) covered by this documentation:

LAMININ_NTER, PS51117; Laminin N-terminal domain profile  (MATRIX)


References

1AuthorsColognato H. Yurchenco P.D.
TitleForm and function: the laminin family of heterotrimers.
SourceDev. Dyn. 218:213-234(2000).
PubMed ID10842354
DOI10.1002/(SICI)1097-0177(200006)218:2<213::AID-DVDY1>3.0.CO;2-R

2AuthorsYurchenco P.D. Cheng Y.S.
TitleSelf-assembly and calcium-binding sites in laminin. A three-arm interaction model.
SourceJ. Biol. Chem. 268:17286-17299(1993).
PubMed ID8349613

3AuthorsXu H. Wu X.R. Wewer U.M. Engvall E.
TitleMurine muscular dystrophy caused by a mutation in the laminin alpha 2 (Lama2) gene.
SourceNat. Genet. 8:297-302(1994).
PubMed ID7874173
DOI10.1038/ng1194-297



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